Los Angeles, a small amount of sperm most often be a scapegoat when a man isdifficult to inseminate his partner. And although polynomial, sperm fertilize the ovum remains difficult because 20 percent of today’s man has a genetic mutation.
Mutations occur in genes DEFB126, which relates to the ability of sperm cells to move closer to the ovum. The gene’s function Formatting carbohydrate coating to help the sperm penetrate the thick mucus that covers the channel to the ovum.
Without a layer of carbohydrate, the sperm will be considered a foreign object so that the mucus-mucus in the vagina will reject its presence. These conditions make the presentdifficult sperm reaching the ovum even though the number and movement of sufficientcapacity.
A study published in the journal Science Translational Medicine recently revealed that 20 percent of today’s men have mutations in genes DEFB126. This finding explains why thecases of infertility in men today tend to increase.
The study, led by Theodore Tollner from the University of California looked at sperm samples donated by 19 volunteers. In experiments in the laboratory, sperm with mutations moves swiftly enough but still failed to penetrate a particular layer.
“We found that sperm cells from donors with mutations have difficulties to penetrate themucosal fluid in the laboratory, are made as closely as possible with mucus,” Tollnersaid as quoted by Reuters.
World Health Organization or WHO estimates, 13-14 infertility problems experienced bycouples and about 50 percent of the problem lies in men. The cause of male infertility is not always known, but the most common scapegoat is the number of sperm that are toosmall.
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